Experts have issued a warning about a new, global healthcare risk.
They are, apparently, extremely worried that an ‘explosive’ increase in new c#vid cases could trigger a “heart failure pandemic”.
MSN reports: Several countries, including the UK, US and China, have seen a surge in C#vid infections over recent weeks, largely due to the influx of a new strain known as JN.1. Health experts are warning this will increase the chances of potential heart issues for those who catch it.
But a new report from scientists at Japan’s top research institute, Riken has warned the ACE2 receptors that cor#navirus clings to within human cells are “very common” in the heart, and many of those who had or have the virus suffer from “reduced cardiac function”.
The reason behind this remains unclear, but the report claims the C#vid pandemic may have significantly changed the situation as those at risk of future heart failure due to “persistent infection of SARS-CoV-2 is expected to exponentially increase”, the Daily Star has reported.
It says: “Even though conclusive clinical evidence that persistent SARS-CoV-2 infection is associated with declined cardiac function has not been reported so far, the proof-of-concept study of the possibility of SARS-CoV-2 persistent infection of the heart and the potential risk of opportunistic progression of heart failure should be validated by a three-dimensional human cardiac tissue model which would serve as the alarm bell for a global healthcare risk.”
Riken Research Leader Hidetoshi Masumoto said: “Some people infected with C#ronavirus may have persistent viral infections in their hearts.
“A testing system and treatment methods must be established in preparation for a ‘heart failure pandemic,’ in which we will see a rapid increase in the number of heart failure patients.
The explosive increase in the number of virus-infected patients due to the C#VID-19 pandemic may have led to an enormous increase in the number of patients at potential risk for future heart failure.
“These patients would be predicted to maintain cardiac function superficially despite being at marginal risk.”